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Christopher Ingalls


Ph. D. in Kinesiology, Texas A&M University, 1994
Master of Science in Kinesiology, Texas A&M University, 1990
Bachelor of Science in Health and Sport Science, Wake Forest University,1988


Skeletal and cardiac muscle physiology and biochemistry:
1) Molecular mechanisms of strength loss and recovery associated with exercise-induced skeletal muscle injury, and
2) Molecular mechanisms of peri-infarct strength loss and cardiac remodeling after myocardial infarction.


Christopher Ingalls has been studying skeletal muscle physiology since 1988. The focus of his research for the last 20 years has been to understand the causes of strength loss and recovery associated with exercise-induced skeletal muscle injury.

To date, his 35 peer-reviewed research papers have been cited over 1,400 times in journal articles and textbooks. For the last 10 years, his research on skeletal muscle physiology and injury in models of human muscle disease has been funded by the National Institutes of Health.

Recently, Ingalls research has expanded to identify causes of cardiac muscle weakness associated with remodeling after myocardial infarction.


Corona, B.T. and C.P. Ingalls. (2013). “Immediate force loss after eccentric contractions is increased with L-NAME administration, a nitric oxide synthase inhibitor”. Muscle & Nerve 47(2):271-273.
Rouviere C., B.T. Corona, and C.P. Ingalls. (2012). “Oxidative Capacity and Fatigability in Run Trained Malignant Hyperthermia Susceptible Mice.” Muscle & Nerve 45(4): 586-596.
Corona, B.T, S.L. Hamilton, and C.P. Ingalls. (2010). “The effect of prior exercise on thermal sensitivity of malignant hyperthermia susceptible muscle.” Muscle & Nerve 42(2): 270-272.
Corona, B.T., E.M. Balog, J.A. Doyle, J.C. Rupp, R.C. Luke, and C.P. Ingalls. (2010). “Junctophilin damage contributes to early strength deficits and EC coupling failure after eccentric contractions.” Am. J . Physiol. Cell Physiol. 298: C365-376.
Corona, B, C. Rouviere, S.L. Hamilton, and C.P. Ingalls. (2008). “FKBP12 deficiency reduces strength deficits after eccentric contraction-induced injury.” J. Appl. Physiol. 105: 527-537.